Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern
Identifieur interne : 000423 ( Main/Corpus ); précédent : 000422; suivant : 000424Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern
Auteurs : Masahito Satoh ; Yoshifusa Aizawa ; Toshikazu Funazaki ; Shinichi Niwano ; Katsuya Ebe ; Seiichi Miyajima ; Kaoru Suzuki ; Masami Aizawa ; Akira ShibataSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1989-03.
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Abstract
In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.
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DOI: 10.1111/j.1540-8159.1989.tb02678.x
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ISTEX:A0ABB100537E586F3366A220C9EEA205071C8AEELe document en format XML
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Japan</mods:affiliation></affiliation></author><author><name sortKey="Suzuki, Kaoru" sort="Suzuki, Kaoru" uniqKey="Suzuki K" first="Kaoru" last="Suzuki">Kaoru Suzuki</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Aizawa, Masami" sort="Aizawa, Masami" uniqKey="Aizawa M" first="Masami" last="Aizawa">Masami Aizawa</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Shibata, Akira" sort="Shibata, Akira" uniqKey="Shibata A" first="Akira" last="Shibata">Akira Shibata</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno 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Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Funazaki, Toshikazu" sort="Funazaki, Toshikazu" uniqKey="Funazaki T" first="Toshikazu" last="Funazaki">Toshikazu Funazaki</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Niwano, Shinichi" sort="Niwano, Shinichi" uniqKey="Niwano S" first="Shinichi" last="Niwano">Shinichi Niwano</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Ebe, Katsuya" sort="Ebe, Katsuya" uniqKey="Ebe K" first="Katsuya" last="Ebe">Katsuya Ebe</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Miyajima, Seiichi" sort="Miyajima, Seiichi" uniqKey="Miyajima S" first="Seiichi" last="Miyajima">Seiichi Miyajima</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Suzuki, Kaoru" sort="Suzuki, Kaoru" uniqKey="Suzuki K" first="Kaoru" last="Suzuki">Kaoru Suzuki</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Aizawa, Masami" sort="Aizawa, Masami" uniqKey="Aizawa M" first="Masami" last="Aizawa">Masami Aizawa</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author><author><name sortKey="Shibata, Akira" sort="Shibata, Akira" uniqKey="Shibata A" first="Akira" last="Shibata">Akira Shibata</name><affiliation><mods:affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Pacing and Clinical Electrophysiology</title><idno type="ISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1989-03">1989-03</date><biblScope unit="volume">12</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="413">413</biblScope><biblScope unit="page" to="420">420</biblScope></imprint><idno type="ISSN">0147-8389</idno></series><idno type="istex">A0ABB100537E586F3366A220C9EEA205071C8AEE</idno><idno type="DOI">10.1111/j.1540-8159.1989.tb02678.x</idno><idno type="ArticleID">PACE413</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0147-8389</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Wolff‐Parkinson‐White syndrome</term><term>atrial fibrillation</term><term>reciprocating tachycardia</term><term>retrograde conduction</term><term>sudden death</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>MASAHITO SATOH</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>YOSHIFUSA AIZAWA</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>TOSHIKAZU FUNAZAKI</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>SHINICHI NIWANO</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>KATSUYA EBE</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>SEIICHI MIYAJIMA</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>KAORU SUZUKI</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>MASAMI AIZAWA</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item><json:item><name>AKIRA SHIBATA</name><affiliations><json:string>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Wolff‐Parkinson‐White syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>reciprocating tachycardia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>atrial fibrillation</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>retrograde conduction</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>sudden death</value></json:item></subject><articleId><json:string>PACE413</json:string></articleId><language><json:string>eng</json:string></language><abstract>In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.</abstract><qualityIndicators><score>6.771</score><pdfVersion>1.6</pdfVersion><pdfPageSize>565 x 792 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>5</keywordCount><abstractCharCount>1257</abstractCharCount><pdfWordCount>3099</pdfWordCount><pdfCharCount>20551</pdfCharCount><pdfPageCount>9</pdfPageCount><abstractWordCount>181</abstractWordCount></qualityIndicators><title>Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title><genre><json:string>article</json:string></genre><host><volume>12</volume><publisherId><json:string>PACE</json:string></publisherId><pages><total>8</total><last>420</last><first>413</first></pages><issn><json:string>0147-8389</json:string></issn><issue>3</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><eissn><json:string>1540-8159</json:string></eissn><title>Pacing and Clinical Electrophysiology</title><doi><json:string>10.1111/(ISSN)1540-8159</json:string></doi></host><publicationDate>1989</publicationDate><copyrightDate>1989</copyrightDate><doi><json:string>10.1111/j.1540-8159.1989.tb02678.x</json:string></doi><id>A0ABB100537E586F3366A220C9EEA205071C8AEE</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/A0ABB100537E586F3366A220C9EEA205071C8AEE/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/A0ABB100537E586F3366A220C9EEA205071C8AEE/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/A0ABB100537E586F3366A220C9EEA205071C8AEE/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><availability><p>WILEY</p></availability><date>1989</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title><author><persName><forename type="first">MASAHITO</forename><surname>SATOH</surname></persName><note type="correspondence"><p>Correspondence: Address for reprints: M. Satoh, M.D., The First Department of Internal Medicine, Niigata University School of Medicine, Asahimachi, Niigata 951, Japan.</p></note><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">YOSHIFUSA</forename><surname>AIZAWA</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">TOSHIKAZU</forename><surname>FUNAZAKI</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">SHINICHI</forename><surname>NIWANO</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">KATSUYA</forename><surname>EBE</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">SEIICHI</forename><surname>MIYAJIMA</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">KAORU</forename><surname>SUZUKI</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">MASAMI</forename><surname>AIZAWA</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author><author><persName><forename type="first">AKIRA</forename><surname>SHIBATA</surname></persName><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation></author></analytic><monogr><title level="j">Pacing and Clinical Electrophysiology</title><idno type="pISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><idno type="DOI">10.1111/(ISSN)1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1989-03"></date><biblScope unit="volume">12</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="413">413</biblScope><biblScope unit="page" to="420">420</biblScope></imprint></monogr><idno type="istex">A0ABB100537E586F3366A220C9EEA205071C8AEE</idno><idno type="DOI">10.1111/j.1540-8159.1989.tb02678.x</idno><idno type="ArticleID">PACE413</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1989</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>Wolff‐Parkinson‐White syndrome</term></item><item><term>reciprocating tachycardia</term></item><item><term>atrial fibrillation</term></item><item><term>retrograde conduction</term></item><item><term>sudden death</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1989-03">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/A0ABB100537E586F3366A220C9EEA205071C8AEE/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="03003"><doi origin="wiley">10.1111/pace.1989.12.issue-3</doi><numberingGroup><numbering type="journalVolume" number="12">12</numbering><numbering type="journalIssue" number="3">3</numbering></numberingGroup><coverDate startDate="1989-03">March 1989</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0041300" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1989.tb02678.x</doi><idGroup><id type="unit" value="PACE413"></id></idGroup><countGroup><count type="pageTotal" number="8"></count></countGroup><titleGroup><title type="tocHeading1">ORIGINAL MANUSCRIPT</title></titleGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="413">413</numbering><numbering type="pageLast" number="420">420</numbering></numberingGroup><correspondenceTo>Address for reprints: M. Satoh, M.D., The First Department of Internal Medicine, Niigata University School of Medicine, Asahimachi, Niigata 951, Japan.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE413.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received October 28, 1988; accepted October 31, 1988.</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="16"></count><count type="linksCrossRef" number="10"></count></countGroup><titleGroup><title type="main">Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>MASAHITO</givenNames><familyName>SATOH</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>YOSHIFUSA</givenNames><familyName>AIZAWA</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>TOSHIKAZU</givenNames><familyName>FUNAZAKI</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>SHINICHI</givenNames><familyName>NIWANO</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>KATSUYA</givenNames><familyName>EBE</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>SEIICHI</givenNames><familyName>MIYAJIMA</familyName></personName></creator><creator creatorRole="author" xml:id="cr7" affiliationRef="#a1"><personName><givenNames>KAORU</givenNames><familyName>SUZUKI</familyName></personName></creator><creator creatorRole="author" xml:id="cr8" affiliationRef="#a1"><personName><givenNames>MASAMI</givenNames><familyName>AIZAWA</familyName></personName></creator><creator creatorRole="author" xml:id="cr9" affiliationRef="#a1"><personName><givenNames>AKIRA</givenNames><familyName>SHIBATA</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="JP"><unparsedAffiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Wolff‐Parkinson‐White syndrome</keyword><keyword xml:id="k2">reciprocating tachycardia</keyword><keyword xml:id="k3">atrial fibrillation</keyword><keyword xml:id="k4">retrograde conduction</keyword><keyword xml:id="k5">sudden death</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="n-fnt-1" numbered="no"><p>This report was partially supported by the Japan Heart Foundation and IBM Japan Research Grant for 1987.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Electrophysiologic Evaluation of Asymptomatic Patients with the Wolff‐Parkinson‐White Pattern</title></titleInfo><name type="personal"><namePart type="given">MASAHITO</namePart><namePart type="family">SATOH</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><description>Correspondence: Address for reprints: M. Satoh, M.D., The First Department of Internal Medicine, Niigata University School of Medicine, Asahimachi, Niigata 951, Japan.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">YOSHIFUSA</namePart><namePart type="family">AIZAWA</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">TOSHIKAZU</namePart><namePart type="family">FUNAZAKI</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">SHINICHI</namePart><namePart type="family">NIWANO</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">KATSUYA</namePart><namePart type="family">EBE</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">SEIICHI</namePart><namePart type="family">MIYAJIMA</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">KAORU</namePart><namePart type="family">SUZUKI</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MASAMI</namePart><namePart type="family">AIZAWA</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">AKIRA</namePart><namePart type="family">SHIBATA</namePart><affiliation>First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1989-03</dateIssued><edition>Received October 28, 1988; accepted October 31, 1988.</edition><copyrightDate encoding="w3cdtf">1989</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">16</extent></physicalDescription><abstract lang="en">In the past 4 years, 34 asymptomatic patients with the Wolff‐Parkinson‐White (WPW) pattern underwent electrophysiologic study. The effective refractory period (ERP) of antegrade conduction over the accessory pathway was 288 ± 29 msec. In three asymptomatic patients (9%), the antegrade ERP of the accessory pathway was shorter than 250 msec. The antegrade ERP of the accessory pathway became shorter than 250 msec in an additional 12 of 22 (55%) patients after isoproterenol administration. Nineteen (56%) of the asymptomatic patients showed the absence of retrograde conduction over the accessory pathway even after isoproterenol administration. The rate of induction of orthodromic reciprocating tachycardia in the asymptomatic WPW patients was 15% (5/34), which was significantly lower than that in the symptomatic patients. These data suggest that in the asymptomatic patients, the absence of retrograde conduction over the accessory pathway is the reason they remained asymptomatic, free of reciprocating tachycardia. However, even in the asymptomatic patients, some had the accessory pathway in which antegrade ERP was shorter than 250 msec. They may result in rapid ventricular conduction over the accessory pathway when atrial fibrillation develops.</abstract><subject lang="en"><genre>Keywords</genre><topic>Wolff‐Parkinson‐White syndrome</topic><topic>reciprocating tachycardia</topic><topic>atrial fibrillation</topic><topic>retrograde conduction</topic><topic>sudden death</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1989</date><detail type="volume"><caption>vol.</caption><number>12</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>413</start><end>420</end><total>8</total></extent></part></relatedItem><identifier type="istex">A0ABB100537E586F3366A220C9EEA205071C8AEE</identifier><identifier type="DOI">10.1111/j.1540-8159.1989.tb02678.x</identifier><identifier type="ArticleID">PACE413</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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